Involved in several processes, including negative regulation of cytokine production; positive regulation of cardiac muscle cell proliferation; and regulation of apoptotic process. Used to study acute myocardial infarction; dilated cardiomyopathy; middle cerebral artery infarction; and myocardial infarction. Biomarker of hepatocellular carcinoma; middle cerebral artery infarction; pre-eclampsia; and transitional cell carcinoma; INTERACTS WITH 17alpha-ethynylestradiol; 2,3,7,8-tetrachlorodibenzodioxine; 5-aza-2'-deoxycytidine.
[INS protein co-treated with Dexamethasone co-treated with 1-Methyl-3-isobutylxanthine co-treated with Indomethacin co-treated with bisphenol A] results in increased expression of H19 mRNA, [INS protein co-treated with Dexamethasone co-treated with 1-Methyl-3-isobutylxanthine co-treated with Indomethacin] results in increased expression of H19 mRNA
Decitabine inhibits the reaction [Smoke results in decreased expression of H19 mRNA], Decitabine inhibits the reaction [tobacco tar results in decreased expression of H19 mRNA]
Decitabine inhibits the reaction [[PDGFB protein co-treated with PDGFB protein] results in decreased expression of H19 mRNA], Decitabine inhibits the reaction [TGFB1 protein results in decreased expression of H19 mRNA]
[INS protein co-treated with Dexamethasone co-treated with 1-Methyl-3-isobutylxanthine co-treated with Indomethacin co-treated with bisphenol A] results in increased expression of H19 mRNA
[INS protein co-treated with Dexamethasone co-treated with 1-Methyl-3-isobutylxanthine co-treated with Indomethacin co-treated with bisphenol A] results in increased expression of H19 mRNA, [INS protein co-treated with Dexamethasone co-treated with 1-Methyl-3-isobutylxanthine co-treated with Indomethacin] results in increased expression of H19 mRNA
[INS protein co-treated with Dexamethasone co-treated with 1-Methyl-3-isobutylxanthine co-treated with Indomethacin co-treated with bisphenol A] results in increased expression of H19 mRNA, [INS protein co-treated with Dexamethasone co-treated with 1-Methyl-3-isobutylxanthine co-treated with Indomethacin] results in increased expression of H19 mRNA
luzindole inhibits the reaction [melatonin inhibits the reaction [hydrogen peroxide decreases expression of H19 RNA in C-kit+ cardiac progenitor cells]]
Metformin co-treated with metformin hydrochloride co-treated with N(gamma)-nitro-L-arginine methyl ester inhibits the reaction [pre-eclampsia increases expression of H19 RNA in peripheral blood]
Diethylnitrosamine promotes the reaction [Carbon Tetrachloride results in increased expression of H19 mRNA], MET protein inhibits the reaction [Diethylnitrosamine results in increased expression of H19 mRNA]
[perfluorooctane sulfonic acid co-treated with Cellulose] results in increased expression of H19 mRNA, [perfluorooctane sulfonic acid co-treated with Pectins] results in increased expression of H19 mRNA
[Plasticizers co-treated with Cosmetics co-treated with Flame Retardants co-treated with perfluorooctanoic acid co-treated with Phytoestrogens] results in decreased expression of H19 mRNA
[Plasticizers co-treated with Cosmetics co-treated with Flame Retardants co-treated with perfluorooctanoic acid co-treated with Phytoestrogens] results in decreased expression of H19 mRNA
H19 overexpression in breast adenocarcinoma stromal cells is associated with tumor values and steroid receptor status but independent of p53 and Ki-67 expression.
Erasure of methylation imprint at the promoter and CTCF-binding site upstream of H19 in human testicular germ cell tumors of adolescents indicate their fetal germ cell origin.
Alterations in promoter usage and expression levels of insulin-like growth factor-II and H19 genes in cervical carcinoma exhibiting biallelic expression of IGF-II.
Changes in the DNA methylation profile of the rat H19 gene upstream region during development and transgenic hepatocarcinogenesis and its role in the imprinted transcriptional regulation of the H19 gene.
Effect of tamoxifen treatment on global and insulin-like growth factor 2-H19 locus-specific DNA methylation in rat spermatozoa and its association with embryo loss.
Metformin exhibits its therapeutic effect in the treatment of pre-eclampsia via modulating the Met/H19/miR-148a-5p/P28 and Met/H19/miR-216-3p/EBI3 signaling pathways.
Phase I/II marker lesion study of intravesical BC-819 DNA plasmid in H19 over expressing superficial bladder cancer refractory to bacillus Calmette-Guerin.
Association of astragaloside IV-inhibited autophagy and mineralization in vascular smooth muscle cells with lncRNA H19 and DUSP5-mediated ERK signaling.
LncRNA H19 promotes the proliferation of pulmonary artery smooth muscle cells through AT1R via sponging let-7b in monocrotaline-induced pulmonary arterial hypertension.
Gene-based anchoring of the rat genetic linkage and cytogenetic maps: new regional localizations, orientation of the linkage groups, and insights into mammalian chromosome evolution.
expression temporally and spatially regulated in the prenatal blood vessel with initial loss of expression in the inner smooth muscle layers adjacent to the lumen
